Bryan Sauls, MD, PhD
Internal Medicine
Practicing At
Accepting New Patients
Yes
Languages Spoken
English
Experience
Practicing since 2008
Medical School
Marshall University
Board Certification(s)
American Board of Internal Medicine
Residency
The Ohio State University Medical Center
Publications
- Lenda, D., B. Sauls, and M. A. Boegehold. Local production of superoxide anion may contribute to reduced arteriolar nitric oxide in rats fed high salt. FASEB J. 13.4:A31, 1999; Sauls, B. A. and M. A. Boegehold.
- A reduction in arteriolar wall PO2 may stimulates nitric oxide release during sympathetic vasoconstriction in the rat intestine. FASEB J. 13.4:A31, 1999; Sauls, B. A. and M. A. Boegehold.
- Local adenosine production may stimulate arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. FASEB J. 14.4:A3, 2000.; Sauls, B. A. and M. A. Boegehold.
- Local PO2 reduction and adenosine release preserve arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. J. Vasc. Res. 37(S1):32, 2000.; Sauls, B. A. and M. A. Boegehold.
- Conversion to adenosine is not responsible for sustained endothelial nitric oxide synthesis during sympathetic vasoconstriction in the rat intestine. FASEB J. 15(4 pt 1):A103, 2001.; Lenda, D. M., B. A. Sauls, and M. A. Boegehold.
- Reactive oxygen species may contribute to reduced endothelium-dependent dilation in rats fed high salt. Am. J. Physiol. Heart Circ. Physiol. 279:H7-H14, 2000.; Sauls, B. A. and M. A. Boegehold.
- Arteriolar wall PO2 and nitric oxide release during sympathetic vasoconstriction in the rat intestine. Am. J. Physiol. Heart Circ. Physiol. 279:H484-H491, 2000.; Sauls, B. A. and M. A. Boegehold.
- Reduced PO2 and adenosine formation preserve arteriolar nitric oxide synthesis during sympathetic constriction in the rat intestine. J. Vasc. Res. 38:104-112, 2001.; Sauls, B. A. and M. A. Boegehold.
- Adenosine linking reduced O2 to arteriolar NO release in intestine is not formed from extracellular ATP. Am. J. Phsyiol. Heart Circ. Physiol. 281:H1193-H1200, 2001"